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Renal toxicity has also been seen in patients in whom renal prostaglandins have a compensatory role in the maintenance of renal perfusionIn these patientsadministration of an NSAID may cause a dose-dependent reduction in prostaglandin formation andsecondarilyin renal blood flowwhich may precipitate overt renal decompensationPatients at greatest risk of this reaction are those with impaired renal functiondehydrationhypovolemiaheart failureliver dysfunctionthose taking diuretics and ACE inhibitors or ARBsand the elderlyDiscontinuation of NSAID therapy is usually followed by recovery to the pretreatment state.
To minimize the potential risk for an adverse CV event in NSAID-treated patientsuse the lowest effective dose for the shortest duration possiblePhysicians and patients should remain alert for the development of such events throughout the entire treatment courseeven in the absence of previous CV symptomsPatients should be informed about the symptoms of serious CV events and the steps to take if they occur.
Renal toxicity has also been seen in patients in whom renal prostaglandins have a compensatory role in the maintenance of renal perfusionIn these patientsadministration of an NSAID may cause a dose-dependent reduction in prostaglandin formation andsecondarilyin renal blood flowwhich may precipitate overt renal decompensationPatients at greatest risk of this reaction are those with impaired renal functiondehydrationhypovolemiaheart failureliver dysfunctionthose taking diuretics and ACE inhibitors or ARBsand the elderlyDiscontinuation of NSAID therapy is usually followed by recovery to the pretreatment state.
This drug passes into breast milk and may have undesirable effects on a nursing infantConsult your doctor before breast-feeding.
All drugs may cause side effectsHowevermany people have no side effects or only have minor side effectsCall your doctor or get medical help if any of these side effects or any other side effects bother you or do not go away:
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